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The Hippo Kinase LATS2 Controls Helicobacter pylori-Induced Epithelial-Mesenchymal Transition and Intestinal Metaplasia in Gastric Mucosa

dc.creatorMolina Castro, Silvia Elena
dc.creatorTiffon, Camille
dc.creatorGiraud, Julie
dc.creatorBoeuf, Hélène
dc.creatorSifré, Elodie
dc.creatorGiese, Alban
dc.creatorBelleannée, Geneviève
dc.creatorLethours, Philippe
dc.creatorBessède, Emilie
dc.creatorMégraud, Francis
dc.creatorDubus, Pierre
dc.creatorStaedel, Cathy
dc.creatorVaron, Christine
dc.date.accessioned2020-12-10T16:24:32Z
dc.date.available2020-12-10T16:24:32Z
dc.date.issued2020
dc.description.abstractGastric carcinoma is related mostly to CagA+-Helicobacter pylori infection, which disrupts the gastric mucosa turnover and elicits an epithelial-mesenchymal transition (EMT) and preneoplastic transdifferentiation. The tumor suppressor Hippo pathway controls stem cell homeostasis; its core, constituted by the large tumor suppressor 2 (LATS2) kinase and its substrate Yes-associated protein 1 (YAP1), was investigated in this context.es
dc.description.procedenceUCR::Vicerrectoría de Investigación::Unidades de Investigación::Ciencias de la Salud::Instituto de Investigaciones en Salud (INISA)es
dc.identifier.citationhttps://www.cmghjournal.org/article/S2352-345X(19)30143-2/fulltext
dc.identifier.doihttps://doi.org/10.1016/j.jcmgh.2019.10.007
dc.identifier.issn2352-345X
dc.identifier.urihttps://hdl.handle.net/10669/82160
dc.language.isoen_US
dc.rightsacceso abierto
dc.sourceCellular and Molecular Gastroenterology and Hepatology 9(2)es
dc.subjectYAP1es
dc.subjectEpithelial-to-Mesenchymal Transitiones
dc.subjectAdenocarcinomaes
dc.subjectCagAes
dc.titleThe Hippo Kinase LATS2 Controls Helicobacter pylori-Induced Epithelial-Mesenchymal Transition and Intestinal Metaplasia in Gastric Mucosaes
dc.typeartículo original

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